Effects of fasudil hydrochloride on delayed cerebral vasospasm after traumatic subarachnoid hemorrhage;
Objective To establish a reliable models of delayed cerebral vasospasm(DCV) after subarachnoid hemorrhage(SAH) in rats.
Results In the angiogram of digital DSA, chronic cerebral vasospasm appeared since the second day after the first cisterna magna injection and reach the peak at the eight day.
Analysis on risk factors of cerebral vasospasm following subarachnoid hemorrhage
Analysis on the risk factors and the therapeutic method of cerebral vasospasm following aneurysmal subarachnoid hemorrhage by interventional therapy
Establishment of cerebral vasospasm model after subarachnoid hemorrhage in pigs
Endothelin and nitric oxide concentration in patients with posttraumatic cerebral vasospasm
Risk factors analysis for cerebral vasospasm after head injury
Dynamic changes of eotaxin-1,nitric oxide in cerebrospinal fluid and their relationships with cerebral vasospasm after skull base tumor resection
Conclusion 1h-mrs can be used to monitor the development of cerebral vasospasm resulting from SAH as a good evaluation method for functional imaging.
Conclusion Naringin can attenuate cerebral vasospasm and decrease the number of neuron apoptosis in brain cortex in rats after SAH, the potential mechanism may be related with anti-oxidant after SAH.
Relationgship between expression of rHSG and delayed cerebral vasospasm after experimental subarachnoid hemorrhage in rats
Effect of simvastatin on vasucular muscle cell proliferation during delayed cerebral vasospasm after subarachnoid hemorrhage.
